Herpes zoster, also known as shingles, develops from reactivation of the varicella zoster virus.1 In immunocompetent individuals, shingles classically presents as pruritic vesicles in a dermatomal distribution with associated constitutional symptoms. The rash typically resolves in 7-10 days.1 The most common complication of herpes zoster is herpetic neuralgia. Motor deficits occur in 1%-5% of patients, with the most common manifestation being Ramsay Hunt Syndrome.2 Other complications include abdominal pseudohernia, localized paresis, constipation, visceral neuropathy, paralytic ileus/colonic pseudo-obstruction, hemidiaphragm paralysis, and bladder dysfunction.