Gonococcal infection primarily affects the mucosal surfaces of the urogenital tract, but rarely causes bacteremia and sepsis due to dissemination1. Disseminated gonococcal infection (DGI) is prevalent in the United States, with a rate of 0.5-3% in patients infected with Neisseria gonorrhoeae2. The main presentation of disseminated gonococcal infection is tenosynovitis, dermatitis, and polyarthralgia, with cutaneous lesions present in 60-90%1. It is critical to have high clinical suspicion of disseminated gonococcal infection, especially in patients presenting with sepsis and immunocompromised status, given the risk of severe complications.