Molecular switch mechanism explains how mutations shorten biological clocks

A new study of molecular interactions central to the functioning of biological clocks explains how certain mutations can shorten clock timing, making some people extreme ‘morning larks’ because their internal clocks operate on a 20-hour cycle instead of being synchronized with the 24-hour cycle of day and night. Researchers found that the same molecular switch mechanism affected by these mutations is at work in animals ranging from fruit flies to people.

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