Gastrointestinal amyloidosis most notably occurs in the liver and small intestine. Its presentation is highly nonspecific and may include gastrointestinal bleeding, malabsorption syndromes, and protein-losing enteropathy. Chronic gastrointestinal dysmotility, while relatively uncommon, carries a particularly grave prognosis. In those with amyloidosis, gastric involvement occurs in 8% of patients, with only 1% being symptomatic.1
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